Mutant A53T alfa-sinüklein, mitokondriyal otofajiyi artırarak nöronal ölümü indükler.
Mutant A53T alpha-synuclein induces neuronal death by increasing mitochondrial autophagy.
DOI: 10.1074/jbc.m110.132514PMID: 21252228
Mutant A53T alpha-synuclein induces neuronal death by increasing mitochondrial autophagy.
DOI: 10.1074/jbc.m110.132514PMID: 21252228
Expression of water-soluble, ligand-binding concatameric extracellular domains of the human neuronal nicotinic receptor alpha4 and beta2 subunits in the yeast Pichia pastoris: glycosylation is not required for ligand binding.
DOI: 10.1074/jbc.m110.171645PMID: 21252231
Leucine-rich repeat kinase 2 and alpha-synuclein: intersecting pathways in the pathogenesis of Parkinson's disease?
DOI: 10.1186/1750-1326-6-6PMID: 21244648
Dispersed activity during passive movement in the globus pallidus of the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated primate.
DOI: 10.1371/journal.pone.0016293PMID: 21267415
The endotoxin-induced neuroinflammation model of Parkinson's disease.
DOI: 10.4061/2011/487450PMID: 21331154
Neuroinflammation and α-synuclein dysfunction potentiate each other, driving chronic progression of neurodegeneration in a mouse model of Parkinson's disease.
DOI: 10.1289/ehp.1003013PMID: 21245015
The biochemical and cellular basis for nutraceutical strategies to attenuate neurodegeneration in Parkinson's disease.
DOI: 10.3390/ijms12010506PMID: 21340000
Mitochondrial Dysfunction: The Road to Alpha-Synuclein Oligomerization in PD.
DOI: 10.4061/2011/693761PMID: 21318163
Enhanced glutamate, IP3 and cAMP activity in the cerebral cortex of unilateral 6-hydroxydopamine induced Parkinson's rats: effect of 5-HT, GABA and bone marrow cell supplementation.
DOI: 10.1186/1423-0127-18-5PMID: 21235809
Microglial MAC1 receptor and PI3K are essential in mediating β-amyloid peptide-induced microglial activation and subsequent neurotoxicity.
DOI: 10.1186/1742-2094-8-3PMID: 21232086
Increased mitochondrial calcium sensitivity and abnormal expression of innate immunity genes precede dopaminergic defects in Pink1-deficient mice.
DOI: 10.1371/journal.pone.0016038PMID: 21249202
Nonmotor Symptoms in Patients with PARK2 Mutations.
DOI: 10.4061/2011/473640PMID: 21317980
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