Scientific Literature Search

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Regenerative Medicine for Parkinson’s Disease.

Yasuhara T, Kameda M, Agari T, Date I.

Neurol Med Chir (Tokyo) · 2015-01-01

DOI: 10.2176/nmc.ra.2014-0264PMID: 26236796

A bibliographic record traceable to Europe PMC. Türkçe başlık: makine çevirisi, editoryal kontrol bekliyor.

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Systemic human CD34(+) cells populate the brain and activate host mechanisms to counteract nigrostriatal degeneration.

Corenblum MJ, Flores AJ, Badowski M, Harris DT, Madhavan L.

Regen Med · 2015-01-01

DOI: 10.2217/rme.15.32PMID: 26237701

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[Parkinson's disease associated with a mutation in the PARK2 gene].

Kaasinen V, Hietala M, Kuoppamäki M.

Duodecim · 2015-01-01

PMID: 26245049

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mTOR Pathway - Novel Modulator of Astrocyte Activity.

Latacz A, Russell JA, Ocłon E, Zubel-łojek J, Pierzchała-Koziec K.

Folia Biol (Krakow) · 2015-01-01

DOI: 10.3409/fb63_2.95PMID: 26255461

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Open access

p38 MAPK and PI3K/AKT Signalling Cascades inParkinson's Disease.

Jha SK, Jha NK, Kar R, Ambasta RK, Kumar P.

Int J Mol Cell Med · 2015-01-01

PMID: 26261796

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Nanoparticles, Neurotoxicity and Neurodegenerative Diseases.

Mushtaq G, Khan JA, Joseph E, Kamal MA.

Curr Drug Metab · 2015-01-01

DOI: 10.2174/1389200216666150812122302PMID: 26264205

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[The use of buspirone in clinical practice].

Levin OS.

Zh Nevrol Psikhiatr Im S S Korsakova · 2015-01-01

DOI: 10.17116/jnevro20151154183-87PMID: 26288293

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Melatonin attenuates MPTP-induced neurotoxicity via preventing CDK5-mediated autophagy and SNCA/α-synuclein aggregation.

Su LY, Li H, Lv L, Feng YM, Li GD, Luo R, Zhou HJ, Lei XG, Ma L, Li JL, Xu L, Hu XT, Yao YG.

Autophagy · 2015-01-01

DOI: 10.1080/15548627.2015.1082020PMID: 26292069

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The Association of Palmitoylethanolamide with Luteolin Decreases Neuroinflammation and Stimulates Autophagy in Parkinson's Disease Model.

Siracusa R, Paterniti I, Impellizzeri D, Cordaro M, Crupi R, Navarra M, Cuzzocrea S, Esposito E.

CNS Neurol Disord Drug Targets · 2015-01-01

DOI: 10.2174/1871527314666150821102823PMID: 26295827

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A Fluorescence Resonance Energy Transfer Assay For Monitoring α- Synclein Aggregation in a Caenorhabditis Elegans Model For Parkinson's Disease.

Nagarajan A, Bodhicharla R, Winter J, Anbalagan C, Morgan K, Searle M, Nazir A, Adenle A, Fineberg A, Brady D, Vere K, Richens J, O'Shea P, Bell D, de-Pomerai D.

CNS Neurol Disord Drug Targets · 2015-01-01

DOI: 10.2174/1871527314666150821110538PMID: 26295817

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Application of Polymeric Nanoparticles for CNS Targeted Zinc Delivery In Vivo.

Chhabra R, Ruozi B, Vilella A, Belletti D, Mangus K, Pfaender S, Sarowar T, Boeckers TM, Zoli M, Forni F, Vandelli MA, Tosi G, Grabrucker AM.

CNS Neurol Disord Drug Targets · 2015-01-01

DOI: 10.2174/1871527314666150821111455PMID: 26295815

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Ion mobility-mass spectrometry-based screening for inhibition of α- synuclein aggregation.

Liu Y, Graetz M, Ho L, Pukala TL.

Eur J Mass Spectrom (Chichester) · 2015-01-01

DOI: 10.1255/ejms.1359PMID: 26307705

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Records come from scientific sources. Bibliographic counts do not represent completed translations or editorial approvals.

191.030 articles found

Regenerative Medicine for Parkinson’s Disease.

Systemic human CD34(+) cells populate the brain and activate host mechanisms to counteract nigrostriatal degeneration.

[Parkinson's disease associated with a mutation in the PARK2 gene].

mTOR Pathway - Novel Modulator of Astrocyte Activity.

p38 MAPK and PI3K/AKT Signalling Cascades inParkinson's Disease.

Nanoparticles, Neurotoxicity and Neurodegenerative Diseases.

[The use of buspirone in clinical practice].

Melatonin attenuates MPTP-induced neurotoxicity via preventing CDK5-mediated autophagy and SNCA/α-synuclein aggregation.

The Association of Palmitoylethanolamide with Luteolin Decreases Neuroinflammation and Stimulates Autophagy in Parkinson's Disease Model.

A Fluorescence Resonance Energy Transfer Assay For Monitoring α- Synclein Aggregation in a Caenorhabditis Elegans Model For Parkinson's Disease.

Application of Polymeric Nanoparticles for CNS Targeted Zinc Delivery In Vivo.

Ion mobility-mass spectrometry-based screening for inhibition of α- synuclein aggregation.

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