NeuralCipher Research overview
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We report the case of a 51-year-old man with lead encephalopathy due to chronic occupational exposure in fishing net manufacturing. Over three years, he developed cognitive decline, cerebellar ataxia, parkinsonism, and myoclonus. MRI showed bilateral hyperintensities T2/FLAIR sequences in deep brain structures, and elevated blood lead levels confirmed the diagnosis [2]. Chelation therapy led to slight improvement in motor symptoms, but cognitive deficits persisted [3]. This case highlights the importance of early recognition and management of lead neurotoxicity to prevent irreversible brain damage.
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